The renin angiotensin-aldosterone system (RAAS) and lipoxins (LXs) have similar roles in many processes. We previously reported that BML-111， a Lipoxin receptor agonist， inhibited chronic injury hepatic fibrosis by regulating RAAS， but whether LXs are involved in BML-111-mediated protection from acute injury is unclear still.
The renin angiotensin-aldosterone system (RAAS) and lipoxins (LXs) have similar roles in many processes. We previously reported that BML-111， a Lipoxin receptor agonist， inhibited chronic injury hepatic fibrosis by regulating RAAS， but whether LXs are involved in BML-111-mediated protection from acute injury is unclear still.
